Clonetics EGM-2 Endothelial Cell Growth Medium-2 is an enhancement of EGM Medium. BBE has been removed and there has been further optimization of the basal media. Final concentration of FBS is 2%.
Clonetics Media Products are specially designed to support the growth of human and animal primary derived cells. Media systems have been specifically developed and optimized for various cell systems. Ys-bio supply.Media are supplied in several formats:
Basal Medium --- Contains no growth factors cytokines, or supplements
SingleQuots Kit --- Growth factors, cytokines, and supplements
BulletKit --- Basal medium and SingleQuots Kit packaged together
EGM Endothelial Cell Growth Medium產(chǎn)品訂購信息：
第一代內(nèi)皮細(xì)胞生長培養(yǎng)基：
LONZA CC-3124 EGM BulletKit (CC-3121 & CC-4133) 咨詢價(jià)格
LONZA CC-3121 EBM Basal Medium 500 ml 咨詢價(jià)格
LONZA CC-3024 EGM Complete Medium 500 ml 咨詢價(jià)格
LONZA CC-3129 EBM Basal Medium Phenol Red Free, 500 ml 咨詢價(jià)格
LONZA CC-4133 EGM SingleQuot Kit Suppl. & Growth Factors 咨詢價(jià)格
EGM-2MV Microvascular Endothelial Cell Growth Medium-2 has been developed to support the growth and expansion of lung microvascular cells. Final concentration of FBS is 5%.Clonetics Media Products are specially designed to support the growth of human and animal primary derived cells. Media systems have been specifically developed and optimized for various cell systems.Ys-bio supply. Media is supplied in several formats:
Basal Medium --- Contains no growth factors cytokines, or supplements
SingleQuots Kit --- Growth factors, cytokines, and supplements
BulletKit --- Basal medium and SingleQuots Kit packaged together
EGM MV Microvascular Endothelial Cell Growth Medium 產(chǎn)品購買信息
LONZA CC-3170 Bronchial Epithelial Cell Growth Medium BEGM支氣管上皮細(xì)胞培養(yǎng)基產(chǎn)品英文簡介
Clonetics Bronchial/Tracheal Epithelial Cell Systems contain Normal Human Bronchial/Tracheal Epithelial Cells (NHBE) or Diseased Human Bronchial/Tracheal Epithelial Cells (DHBE) and optimized media for their growth. Each System can quickly generate NHBE or DHBE cultures for experimental applications in cancer research, respiratory disease, cellular function and differentiation. Clonetics Bronchial/Tracheal Epithelial Cell Systems are convenient and easy to use, allowing the researcher to focus on results. There are three types of DHBE available cells isolated from donors with cystic fibrosis, asthma, and COPD. Cryopreserved NHBE and DHBE are shipped as frozen primaries. Proliferating NHBE and DHBE are shipped in second passage. Clonetics Cells, Medium and Reagents are quality tested together and guaranteed to give optimum performance as a complete Cell System.Ys-bio supply.Clonetics Media Products are specially designed to support the growth of human and animal primary derived cells. Media systems have been specifically developed and optimized for various cell systems. BEGM BulletKit is serum free and has been optimized to grow diseased and normal bronchial epithelial cells.
1. Al-Wadei HAN, Takahashi T, Schuller M.Growth stimulation of human pulmonary adenocarcinoma cells and small airway
epithelial cells by ß-carotene via activation of cAMP, PKA, CREB, and ERK1/2. Int J Canc. 2006; 118(6):1370-80.
2. Ashitani J, Kyoraku Y, Yanagi S, Matsumoto N, Nakazato M. Elevated levels of beta-Dglucan in bronchoalveolar lavage fluid in
patients with farmer's lung in Miyazaki,Japan. Respiration. 2008; 75(2):182-8. 3. Avadhanula V, Rodriguez CA, DeVincenzoJP, Wang Y, Webby RJ, Ulett GC, Adderson EE. Respiratory viruses augment the adhesion of bacterial pathogens to respiratory epithelium in a viral species- and cell-type dependent manner. J Virol. 2006 Feb; 80(4):1629-36.
4. Chang GC, Liu KJ, Hsieh CL, Hu TS, Charoenfuprasert S, Liu HK, Luh KT, HsuLH, Wu CW, Ting CC, Chen CY, Chen KC,
Yang TY, Chou TY, Wang WH, WhangPeng J, Shih NY. Identification of alphaenolase as an autoantigen in lung cancer: its overexpression is associated with clinical outcomes. Clin Cancer Res. 2006; 12(19):5746-54.
5. Douillet CD, Robinson WP,Milano PM, Boucher RC, Rich PB. Nucleotides induce IL-6 release from human airway epithelia via
P2Y2 and p38 MAPK-dependent pathways. Am J Physiol Lung Cell Mol Physiol. 2006; 291(4):L734-46.
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